IBL/TNF-α（TNF-a）高灵敏度ELISA/BE58351/
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产品分类：
夹心法ELISA
公司分类：
Sandwich_method_ELISA
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3392242852
电话号码：
4000-520-616
电子邮箱：
info@ebiomall.com
商品介绍
| Kitsize | 12x8 |
| Method | ELISA |
| Incubationtime | 1x2h,1x1h,1x15min,1x30min,1x10-20min |
| Standardrange | 0.31-20pg/ml |
| Specimen/Volumes | 50µlserum,plasma,cellculturesupernatant |
| Substrate/isotope | TMB450nm |
| RegulatoryStatus: | Forresearchuseonly |
Detailsfor:TNF-alpha(TNF-a)HighSensitivityELISA
ThisHighSensitivityELISAenablestheanalysisofTNF-alphainlowconcentrationsfromserum,plasma,orcellculturesamples.TheHighSensitivityELISAKitsofferaccuratequantificationbelowconventionalELISAlimits.
TNF-alphareleaseismainlytriggeredbyviralinfections,endotoxins,lipopolysaccharidesorotherbacterialcomponents,bytissueinjury,DNA-damageandbyIL-1,PDFGandTNF-alphaitself.Itisprimarilyexpressedinmacrophages,butalsoinmonocytes,neutrophils,NK-cells,mast-cells,endothelialcellsandactivatedlymphocytes.TNF-alphaexpressioninendothelialcellsandfibroblastscanbeinducedbyIL-17.Theexpressionofothercytokines,chemokines,reactiveoxygenintermediates,nitricoxideandprostaglandinsisstimulatedbyTNF-alpha.MultipleBIOLOGicalfunctionsofTNF-alphacomprisecellularproliferationanddifferentiation,tumOrigenesis,apoptoticornecroticcelldeath(includingcertaintumorcelllines),immunoregulatoryactivities,lipidmetabolism,coagulationandendothelialfunction.Itpromoteslocalorsystemicinflammation(TNF-alphaisapotentpyrogen)andstimulatestheacutephaseresponse.VeryhighexpressionsofTNF-alphaafterinfectioncanleadtosepticshock(TNF-alphaishighlycytotoxic.),whereassustainedlowlevelsinducecachexiaandinflammation.DysregulationofTNF-alphaisinvolvedinmanydiseases.
Synonyms:Cachectin;TNFligandsuperfamilymember2
ForconcretedatapleaseconsulttheInstructionforUseinthedownloadboxontherightside.TNF-alphareleaseismainlytriggeredbyviralinfections,endotoxins,lipopolysaccharidesorotherbacterialcomponents,bytissueinjury,DNA-damageandbyIL-1,PDFGandTNF-alphaitself.Itisprimarilyexpressedinmacrophages,butalsoinmonocytes,neutrophils,NK-cells,mast-cells,endothelialcellsandactivatedlymphocytes.TNF-alphaexpressioninendothelialcellsandfibroblastscanbeinducedbyIL-17.Theexpressionofothercytokines,chemokines,reactiveoxygenintermediates,nitricoxideandprostaglandinsisstimulatedbyTNF-alpha.MultipleBIOLOGicalfunctionsofTNF-alphacomprisecellularproliferationanddifferentiation,tumOrigenesis,apoptoticornecroticcelldeath(includingcertaintumorcelllines),immunoregulatoryactivities,lipidmetabolism,coagulationandendothelialfunction.Itpromoteslocalorsystemicinflammation(TNF-alphaisapotentpyrogen)andstimulatestheacutephaseresponse.VeryhighexpressionsofTNF-alphaafterinfectioncanleadtosepticshock(TNF-alphaishighlycytotoxic.),whereassustainedlowlevelsinducecachexiaandinflammation.DysregulationofTNF-alphaisinvolvedinmanydiseases.
Synonyms:Cachectin;TNFligandsuperfamilymember2
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