IBL/GIP活性高灵敏度（小鼠）ELISA/30116203/
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美元价：
0.00
产品分类：
夹心法ELISA
公司分类：
Sandwich_method_ELISA
联系Q Q：
3392242852
电话号码：
4000-520-616
电子邮箱：
info@ebiomall.com
商品介绍
| Kit size | 12 x 8 |
| Method | ELISA |
| Incubation time | 1 x 18h, 1 x 1h, 1 x 30 min. |
| Standard range | 0.5 - 32 pmol/L |
| Specimen / Volumes | 5 µL plasma |
| Substrate / isotope | TMB 450 nm |
| Regulatory Status: | For research use only |
Details for:
GIP active High Sensitive (mouse) ELISA
"Original article #27702
Incretins are a group of gastrointestinal hormones that cause an increase in the amount of insulin released from beta cells of the pancreatic islets after food intake and they also inhibit glucagon release from the alpha cells of pancreatic islets. GIP, another typical incretin like GLP-1, was isolated and sequenced from intestinal mucosa as “gastric inhibitory peptide” in 1970, and then it was renamed as “glucose-dependent insulinotropic peptide”. It has been shown that GIP receptor is expressed in cells such as pancreatic beta cells, adipocytes or osteoblastic cells, and it plays essential roles in retaining ingested nutrients within the body in each cell, and the control of GIP signaling can lead to improvement of metabolic syndrome. It is rapidly inactivated to GIP (3-42) from active form of GIP (1-42) by DPP-IV in blood. This ELISA kit can measure only active form of Mouse GIP (1-42)."
Separate control set available on request
For concrete data please consult the Instruction for Use in the download box on the right side. Incretins are a group of gastrointestinal hormones that cause an increase in the amount of insulin released from beta cells of the pancreatic islets after food intake and they also inhibit glucagon release from the alpha cells of pancreatic islets. GIP, another typical incretin like GLP-1, was isolated and sequenced from intestinal mucosa as “gastric inhibitory peptide” in 1970, and then it was renamed as “glucose-dependent insulinotropic peptide”. It has been shown that GIP receptor is expressed in cells such as pancreatic beta cells, adipocytes or osteoblastic cells, and it plays essential roles in retaining ingested nutrients within the body in each cell, and the control of GIP signaling can lead to improvement of metabolic syndrome. It is rapidly inactivated to GIP (3-42) from active form of GIP (1-42) by DPP-IV in blood. This ELISA kit can measure only active form of Mouse GIP (1-42)."
Separate control set available on request
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